BARBITURATES IN PRIMATE CORTEX FOCAL ISCHEMIA/Branston
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چکیده
19. Rampton DS, Ramsay DJ: The effects of pentobarbitone anaesthesia on the volume and composition of the extracellular fluid of dogs. J Physiol (Lond) 237: 521-533, 1974 20. Astrup J, Nordstrom C-H, Rehncrona S: Rate of rise in extracellular potassium in the ischemic rat brain and the effect of pre-ischemic metabolic rate: Evidence for a specific effect of phenobarbitone. In Ingvar DH, Lassen NA (eds) Cerebral Function, Metabolism and Circulation. Copenhagen, Munksgaard. pp 7.8-7.9, 1977 21. Guiditta A: The inhibitions of a brain diaphorase by some hypnotics and nervous depressants. J Neurochem 9: 329-336, 1962 22. Guiditta A, Di Prisco G: The inhibitions of cerebral NAD(P)H: (acceptor) oxido-reductase by barbiturates and nervous depressants. Relationship with chemical structure. Biochem Biophys Acta 77: 394-407, 1963 23. Symon L, Branston NM, Chikovani 0: Ischemic brain edema following middle cerebral artery occlusion in baboons: Relationship between regional cerebral water content and blood flow at 1-2 hours. Stroke 10: 184-191, 1979 24. Symon L, Crockard HA, Dorsch NWC et al: Local cerebral blood flow and vascular reactivity in a chronic stable stroke in baboons. Stroke 6: 482-492, 1975
منابع مشابه
Barbiturates in focal ischemia of primate cortex: effects on blood flow distribution, evoked potential and extracellular potassium.
brain ischemia: protection from irreversible damage with a rapid acting barbiturate (Methohexital). Stroke 3: 726-732, 1972 11. Clasen R, Pandolfi S, Casey D: Furosemide and pentobarbital in cryogenic cerebral injury and edema. Neurology (Minneap) 24: 642-648, 1974 12. Smith A, Marque J: Anesthetics and cerebral edema. Anesthesiology 45: 64-72, 1976 13. Anderson D, Crawford R: Corticosteroids i...
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تاریخ انتشار 2005